TY - JOUR
AU - Rothemann, Robin Alexander
AU - Pavlenko, Egor
AU - Mondal, Mrityunjoy
AU - Gerlich, Sarah
AU - Grobushkin, Pavel
AU - Mostert, Sebastian
AU - Racho, Julia
AU - Weiss, Konstantin
AU - Stobbe, Dylan
AU - Stillger, Katharina
AU - Lapacz, Kim
AU - Salscheider, Silja Lucia
AU - Petrungaro, Carmelina
AU - Ehninger, Dan
AU - Nguyen, Thi Hoang Duong
AU - Dengjel, Jörn
AU - Neundorf, Ines
AU - Bano, Daniele
AU - Poepsel, Simon
AU - Riemer, Jan
TI - Interaction with AK2A links AIFM1 to cellular energy metabolism.
JO - Molecular cell
VL - 85
IS - 13
SN - 1097-2765
CY - [Cambridge, Mass.]
PB - Cell Press
M1 - DZNE-2025-00773
SP - 2550 - 2566.e6
PY - 2025
AB - Apoptosis-inducing factor 1 (AIFM1) is a flavoprotein essential for mitochondrial function and biogenesis. Its interaction with MIA40/CHCHD4, the central component of the mitochondrial disulfide relay, accounts for some, but not all, aspects of AIFM1 function. We provide a high-confidence AIFM1 interactome that elucidates functional partners within the mitochondrial intermembrane space. We found that AIFM1 binding to adenylate kinase 2 (AK2), an essential enzyme that maintains cellular adenine nucleotide pools, depends on the AK2 C-terminal domain. High-resolution cryoelectron microscopy (cryo-EM) and biochemical analyses showed that both MIA40 and AK2A bind the AIFM1 C-terminal β-sheet domain. Their binding enhances NADH oxidoreductase activity by locking an active dimer conformation and, in the case of MIA40, affecting the cofactor-binding site. The AIFM1-AK2A interaction is important during mitochondrial respiration because AIFM1 serves as a recruiting hub within the IMS, regulating mitochondrial bioenergetic output by creating hotspots of metabolic enzymes.
KW - AIFM1 (Other)
KW - AK2 (Other)
KW - ATP (Other)
KW - ATP transport (Other)
KW - MIA40/CHCHD4 (Other)
KW - MICOS (Other)
KW - metabolism (Other)
KW - mitochondria (Other)
LB - PUB:(DE-HGF)16
C6 - pmid:40578348
DO - DOI:10.1016/j.molcel.2025.05.036
UR - https://pub.dzne.de/record/279442
ER -