000280043 001__ 280043 000280043 005__ 20250824001723.0 000280043 0247_ $$2doi$$a10.1038/s41467-025-61650-z 000280043 0247_ $$2pmid$$apmid:40691194 000280043 0247_ $$2altmetric$$aaltmetric:179543192 000280043 037__ $$aDZNE-2025-00882 000280043 041__ $$aEnglish 000280043 082__ $$a500 000280043 1001_ $$00000-0002-0531-6309$$aBracher-Smith, Matthew$$b0 000280043 245__ $$aMachine learning in Alzheimer's disease genetics. 000280043 260__ $$a[London]$$bSpringer Nature$$c2025 000280043 3367_ $$2DRIVER$$aarticle 000280043 3367_ $$2DataCite$$aOutput Types/Journal article 000280043 3367_ $$0PUB:(DE-HGF)16$$2PUB:(DE-HGF)$$aJournal Article$$bjournal$$mjournal$$s1753188759_31092 000280043 3367_ $$2BibTeX$$aARTICLE 000280043 3367_ $$2ORCID$$aJOURNAL_ARTICLE 000280043 3367_ $$00$$2EndNote$$aJournal Article 000280043 520__ $$aTraditional statistical approaches have advanced our understanding of the genetics of complex diseases, yet are limited to linear additive models. Here we applied machine learning (ML) to genome-wide data from 41,686 individuals in the largest European consortium on Alzheimer's disease (AD) to investigate the effectiveness of various ML algorithms in replicating known findings, discovering novel loci, and predicting individuals at risk. We utilised Gradient Boosting Machines (GBMs), biological pathway-informed Neural Networks (NNs), and Model-based Multifactor Dimensionality Reduction (MB-MDR) models. ML approaches successfully captured all genome-wide significant genetic variants identified in the training set and 22% of associations from larger meta-analyses. They highlight 6 novel loci which replicate in an external dataset, including variants which map to ARHGAP25, LY6H, COG7, SOD1 and ZNF597. They further identify novel association in AP4E1, refining the genetic landscape of the known SPPL2A locus. Our results demonstrate that machine learning methods can achieve predictive performance comparable to classical approaches in genetic epidemiology and have the potential to uncover novel loci that remain undetected by traditional GWAS. These insights provide a complementary avenue for advancing the understanding of AD genetics. 000280043 536__ $$0G:(DE-HGF)POF4-353$$a353 - Clinical and Health Care Research (POF4-353)$$cPOF4-353$$fPOF IV$$x0 000280043 588__ $$aDataset connected to CrossRef, PubMed, , Journals: pub.dzne.de 000280043 650_7 $$2NLM Chemicals$$aGTPase-Activating Proteins 000280043 650_2 $$2MeSH$$aAlzheimer Disease: genetics 000280043 650_2 $$2MeSH$$aHumans 000280043 650_2 $$2MeSH$$aMachine Learning 000280043 650_2 $$2MeSH$$aGenome-Wide Association Study 000280043 650_2 $$2MeSH$$aGenetic Predisposition to Disease 000280043 650_2 $$2MeSH$$aPolymorphism, Single Nucleotide 000280043 650_2 $$2MeSH$$aAlgorithms 000280043 650_2 $$2MeSH$$aGTPase-Activating Proteins: genetics 000280043 650_2 $$2MeSH$$aNeural Networks, Computer 000280043 7001_ $$aMelograna, Federico$$b1 000280043 7001_ $$aUlm, Brittany$$b2 000280043 7001_ $$00000-0002-1240-7874$$aBellenguez, Céline$$b3 000280043 7001_ $$aGrenier-Boley, Benjamin$$b4 000280043 7001_ $$aDuroux, Diane$$b5 000280043 7001_ $$aNevado, Alejo J$$b6 000280043 7001_ $$00000-0003-0870-9412$$aHolmans, Peter$$b7 000280043 7001_ $$00000-0002-2612-1797$$aTijms, Betty M$$b8 000280043 7001_ $$00000-0002-9889-3606$$aHulsman, Marc$$b9 000280043 7001_ $$00000-0002-2148-381X$$ade Rojas, Itziar$$b10 000280043 7001_ $$00000-0002-1395-8571$$aCampos-Martin, Rafael$$b11 000280043 7001_ $$00000-0003-1606-8643$$ader Lee, Sven van$$b12 000280043 7001_ $$aCastillo, Atahualpa$$b13 000280043 7001_ $$00000-0002-3835-9639$$aKüçükali, Fahri$$b14 000280043 7001_ $$0P:(DE-2719)2811024$$aPeters, Oliver$$b15 000280043 7001_ $$0P:(DE-2719)2812035$$aSchneider, Anja$$b16 000280043 7001_ $$0P:(DE-2719)2000030$$aDichgans, Martin$$b17 000280043 7001_ $$00000-0002-1432-313X$$aRujescu, Dan$$b18 000280043 7001_ $$00000-0003-1759-6990$$aScherbaum, Norbert$$b19 000280043 7001_ $$aDeckert, Jürgen$$b20 000280043 7001_ $$00000-0003-4321-6090$$aRiedel-Heller, Steffi$$b21 000280043 7001_ 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