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@ARTICLE{Wiora:280246,
author = {Wiora, Linus and Yuan, Qinggong and Hook, Sebastian and
Kraft, Melanie and Björkhem, Ingemar and Ott, Michael and
Hauser, Stefan and Schöls, Ludger},
title = {{AAV}8-based gene replacement therapy for hereditary
spastic paraplegia type 5.},
journal = {Molecular therapy / Methods $\&$ clinical development},
volume = {33},
number = {3},
issn = {2329-0501},
address = {New York, NY},
publisher = {Nature Publishing Group},
reportid = {DZNE-2025-00924},
pages = {101531},
year = {2025},
abstract = {Hereditary spastic paraplegia type 5 (SPG5) is an autosomal
recessive neurological disorder caused by mutations in the
CYP7B1 gene, which encodes cholesterol 7α-hydroxylase, an
essential enzyme in cholesterol metabolism. These mutations
lead to elevated levels of 25- and 27-hydroxycholesterol,
oxysterols known to be neurotoxic and blood-brain-barrier
permeable. Their accumulation contributes significantly to
SPG5 pathogenesis, resulting in spastic gait disturbance and
severely impaired quality of life. Using a Cyp7b1-/- mouse
model that mirrors the metabolic phenotype of SPG5, we
developed a gene therapy approach to correct oxysterol
imbalance. We designed an AAV8-TTR-hCYP7B1 vector to deliver
the CYP7B1 gene specifically to the liver. Following
intravenous administration, oxysterol levels in blood and
liver were rapidly normalized, even at low doses (1E10),
with no observed toxicity at the highest tested dose (1E11).
Despite these promising peripheral results, oxysterol levels
in the brain, particularly 27-hydroxycholesterol, remained
only partially corrected six weeks post-treatment. Our
findings suggest that while liver-targeted gene therapy is
effective at restoring peripheral cholesterol metabolism, a
successful therapeutic strategy for SPG5 must also address
central nervous system involvement. We conclude that
successful treatment of SPG5 would require a novel gene
therapeutic approach that also targets the CNS.},
keywords = {AAV (Other) / CYP7B1 (Other) / SPG5 (Other) / cholesterol
(Other) / gene therapy (Other) / hereditary spastic
paraplegia (Other) / neurodegeneration (Other) / oxysterols
(Other)},
cin = {AG Schöls / AG Hauser},
ddc = {610},
cid = {I:(DE-2719)5000005 / I:(DE-2719)1210016},
pnm = {353 - Clinical and Health Care Research (POF4-353) / 352 -
Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-353 / G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40741602},
pmc = {pmc:PMC12309954},
doi = {10.1016/j.omtm.2025.101531},
url = {https://pub.dzne.de/record/280246},
}