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@ARTICLE{Otto:280918,
author = {Otto, Johannes and Statz, Meike and Weber, Hanna and
Koschay, Maximilian and Kober, Maria and Plocksties, Franz
and Timmermann, Dirk and Haubelt, Christian and Storch,
Alexander and Fauser, Mareike and Grützmacher, Florian and
Spors, Sascha},
title = {{A}ccelerometry is a valid method to distinguish between
healthy and 6-{OHDA}-lesioned parkinsonian rats.},
journal = {Scientific reports},
volume = {15},
number = {1},
issn = {2045-2322},
address = {[London]},
publisher = {Springer Nature},
reportid = {DZNE-2025-01001},
pages = {31883},
year = {2025},
abstract = {In Parkinson's disease (PD), continuous sensor-based
evaluation of motor symptom severity, e.g., using
accelerometry, has become an emerging field of interest in
clinical research. Continuous symptom monitoring would also
be of interest in preclinical disease models; however, such
devices are far less established in animal models, most
likely due to additional requirements in size, energy
consumption, and impairment-free attachment. In contrast,
accelerometers manufactured in micro-electro-mechanical
systems (MEMS) technology are promising sensor devices,
which allow for space-saving and energy-efficient monitoring
of movements. In the present study, we aim to extend the
state of the art by establishing wireless accelerometer
measurements as a simple and energy-efficient method to
distinguish between healthy rats and the 6-hydroxydopamine
(6-OHDA) PD animal model. Male Wistar-Han rats were assessed
either three weeks after unilateral 6-OHDA or sham lesioning
within their home cages with an extracorporeal accelerometer
placed in a rodent backpack for 12 h during their active
phase. The data was transmitted wirelessly to a computer,
preprocessed, and a statistical analysis was performed to
find differences between the datasets of 6-OHDA and
sham-lesioned rats. The statistical analysis showed
significant differences in the variances of the magnitude of
the acceleration vectors between the two classes. In
conclusion, accelerometry is a valid method to distinguish
between 6-OHDA-lesioned rats with unilateral dopaminergic
deficiency and their healthy counterparts. The presented
method represents a first step towards automated symptom
severity monitoring and provides a framework to expand the
application to on-implant integrated accelerometers for
continuous monitoring of symptom manifestations in rodent
models of neurodegenerative diseases. Future studies are
required to expand accelerometry to assess symptom severity
to ultimately utilize it for preclinical research on
adaptive therapies.},
keywords = {Animals / Accelerometry: methods / Accelerometry:
instrumentation / Oxidopamine: toxicity / Rats / Male /
Disease Models, Animal / Parkinson Disease: diagnosis /
Parkinson Disease: physiopathology / Rats, Wistar / Wireless
Technology / 6-hydroxydopamine (Other) / Accelerometry
(Other) / Biomarkers (Other) / MEMS accelerometer (Other) /
Parkinson’s disease (Other) / Oxidopamine (NLM Chemicals)},
cin = {AG Storch},
ddc = {600},
cid = {I:(DE-2719)5000014},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40883378},
pmc = {pmc:PMC12397390},
doi = {10.1038/s41598-025-17278-6},
url = {https://pub.dzne.de/record/280918},
}