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@INBOOK{Hashkes:280929,
      author       = {Saavedra, Victor and Moghaddas, Fiona and Latz, Eicke and
                      Masters, Seth L.},
      editor       = {Hashkes, Philip J. and Laxer, Ronald M. and Simon, Anna},
      title        = {{P}attern {R}ecognition {R}eceptors in {A}utoinflammation},
      address      = {Cham},
      publisher    = {Springer International Publishing},
      reportid     = {DZNE-2025-01012},
      pages        = {61 - 87},
      year         = {2019},
      comment      = {Textbook of Autoinflammation / Hashkes, Philip J. (Editor)
                      ; Cham : Springer International Publishing, 2019, Chapter 4
                      ; ISBN: 978-3-319-98604-3 ; doi:10.1007/978-3-319-98605-0},
      booktitle     = {Textbook of Autoinflammation /
                       Hashkes, Philip J. (Editor) ; Cham :
                       Springer International Publishing,
                       2019, Chapter 4 ; ISBN:
                       978-3-319-98604-3 ;
                       doi:10.1007/978-3-319-98605-0},
      abstract     = {The immune system is essential for maintenance of tissue
                      homeostasis. This task requires that immune cells detect and
                      respond to dyshomeostatic states (when homeostasis has
                      broken down) that can occur during invasion of the host with
                      pathogenic microbes, after sterile trauma of tissues or
                      during metabolic derangements. Research in the field of
                      innate immunity has uncovered many molecular mechanisms by
                      which the immune system can prevent the spread of infection,
                      restore damaged tissues and respond to altered metabolism.
                      These pathways involve different classes of pattern
                      recognition receptors, some of which can directly detect
                      minimal motifs (patterns) that are common to multiple
                      pathogens or types of damaged cells. Here, we summarize the
                      general concepts that have been developed to explain how
                      immune recognition of dyshomeostasis is achieved and discuss
                      our current knowledge of the innate immune signaling
                      receptors that are known to directly bind ligands.},
      cin          = {AG Latz},
      cid          = {I:(DE-2719)1013024},
      pnm          = {351 - Brain Function (POF4-351)},
      pid          = {G:(DE-HGF)POF4-351},
      typ          = {PUB:(DE-HGF)7},
      doi          = {10.1007/978-3-319-98605-0_4},
      url          = {https://pub.dzne.de/record/280929},
}