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@ARTICLE{Lo:280963,
author = {Lo, Hung and Riedel, Walter Cañedo and Tantirigama,
Malinda L S and Schoenherr, Anke and Moreno Velasquez, Laura
and Faiss, Lukas and Kumar, Amit and Hakus, Aileen and Rost,
Benjamin R and Larkum, Matthew E and Judkewitz, Benjamin and
Stumpenhorst, Katharina and Rivalan, Marion and Winter, York
and Russo, Eleonora and Kelsch, Wolfgang and Schmitz,
Dietmar and Johenning, Friedrich W},
title = {{F}eeding-induced olfactory cortex suppression reduces
satiation.},
journal = {Neuron},
volume = {113},
number = {17},
issn = {0896-6273},
address = {[Cambridge, Mass.]},
publisher = {Cell Press},
reportid = {DZNE-2025-01045},
pages = {2856-2871.e8},
year = {2025},
abstract = {Binge feeding commonly leads to overeating. Experiencing
flavor during food consumption contributes to satiation.
Still, the interactions between flavor, binge feeding, and
food intake remain unknown. Using miniscopes for in vivo
calcium imaging in the anterior piriform cortex (aPC) in
freely moving mice, we identified specific excitatory
neuronal responses to different food flavors during slow
feeding. Switching from slow feeding to binge feeding
transformed these specific responses into an unspecific
global suppression of neuronal activity. Consummatory aPC
suppression scaled with food value. GABAergic neurons in the
olfactory tubercle (OT) projected to the aPC and mirrored
activity patterns in the aPC under different feeding
conditions, consistent with transmitting a value signal.
Closed-loop optogenetic manipulations demonstrated that
suppressing the aPC during binge bouts reduces satiation by
selectively prolonging feeding bouts. We propose that aPC
suppression by the OT enhances food intake by reducing
sensory satiation during binge feeding-associated states of
high motivation.},
keywords = {Ca(2+) imaging (Other) / binge eating (Other) / consumption
(Other) / flavor (Other) / functional connectivity (Other) /
olfactory tubercle (Other) / optogenetics (Other) / piriform
cortex (Other) / sensory satiation (Other) / smell (Other)},
cin = {AG Schmitz},
ddc = {610},
cid = {I:(DE-2719)1810004},
pnm = {351 - Brain Function (POF4-351)},
pid = {G:(DE-HGF)POF4-351},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40818450},
doi = {10.1016/j.neuron.2025.07.020},
url = {https://pub.dzne.de/record/280963},
}