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@ARTICLE{Dowling:280973,
author = {Dowling, Paul and Negroni, Elisa and Trollet, Capucine and
Zweyer, Margit and Swandulla, Dieter and Ohlendieck, Kay},
title = {{S}erum protein biomarker signature of {D}uchenne muscular
dystrophy.},
journal = {European journal of translational myology},
volume = {35},
number = {2},
issn = {2037-7452},
address = {Padova},
publisher = {Unipress},
reportid = {DZNE-2025-01055},
pages = {13956},
year = {2025},
abstract = {In contrast to invasive skeletal muscle biopsies and the
associated complexity of tissue sampling techniques and
potential detrimental side effects, the alternative
application of liquid biopsy procedures has considerable
advantages concerning minimal invasiveness, repeated
sampling options, assay robustness and cost effectiveness.
This article outlines the current status of serum biomarkers
used for diagnosing and characterizing Duchenne muscular
dystrophy (DMD), a primary muscle wasting disease of early
childhood due to primary abnormalities in the extremely
large DMD gene. Reviewed are important aspects of the
discovery, characterization and diagnostic value of
biofluid-based protein markers of dystrophinopathy. This
includes an overview of traditional general skeletal muscle
damage markers, such as creatine kinase, myoglobin and
lactate dehydrogenase, which have been used for many decades
in clinical applications to evaluate patients with muscular
weakness. In addition, this article outlines the biochemical
identification of novel biomarker candidates focusing on the
usage of mass spectrometry-based proteomic surveys to
establish comprehensive profiles of protein alterations in
dystrophinopathy. Pathoproteomic serum markers of
myonecrosis with great potential for improved patient
screening, differential diagnosis, stage-specific prognosis
and therapeutic monitoring include specific isoforms of
muscle-derived cytosolic proteins, such as carbonic
anhydrase isoform CA3 and fatty acid binding protein FABP3,
as well as sarcomeric proteins, including specific isoforms
of myosin light chain, myosin binding protein, troponin, and
myomesin, in addition to peptide fragments derived from the
giant protein titin. Biofluid-associated marker proteins of
reactive myofibrosis include the extracellular matrix
proteins fibronectin, osteopontin, collagen and
matrix-metalloproteinases.},
cin = {AG Salomoni},
ddc = {610},
cid = {I:(DE-2719)1013032},
pnm = {352 - Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40438995},
pmc = {pmc:PMC12265423},
doi = {10.4081/ejtm.2025.13956},
url = {https://pub.dzne.de/record/280973},
}