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@ARTICLE{Imtiaz:281276,
author = {Imtiaz, Mohammed Aslam and Melas, Konstantinos and Tin,
Adrienne and Talevi, Valentina and Chen, Honglei and
Fornage, Myriam and Shrestha, Srishti and Gögele, Martin
and Emmert, David and Pattaro, Cristian and Pramstaller,
Peter and Förster, Franz and Horn, Katrin and Mosley,
Thomas H and Fuchsberger, Christian and Scholz, Markus and
Breteler, Monique M B and Aziz, N. Ahmad},
title = {{G}enome-wide association study meta-analysis uncovers
novel genetic variants associated with olfactory
dysfunction.},
journal = {BMC genomic data},
volume = {26},
number = {1},
issn = {2730-6844},
address = {[London]},
publisher = {BioMed Central},
reportid = {DZNE-2025-01091},
pages = {64},
year = {2025},
abstract = {Olfactory dysfunction is among the earliest signs of many
age-related neurodegenerative diseases and has been
associated with increased mortality in older adults;
however, its genetic basis remains largely unknown.
Therefore, here we aimed to elucidate its genetic
architecture through a genome-wide association study
meta-analysis (GWMA).This GWMA included the participants of
European ancestry (N = 22,730) enrolled in four different
large population-based studies followed by a multi-ancestry
GWMA including participants of African ancestry (N = 1,030).
Olfactory dysfunction was assessed using a 12-item smell
identification test.GWMA revealed a novel genome-wide
significant locus (tagged by single nucleotide polymorphism
rs11228623 at the 11q12 locus) associated with olfactory
dysfunction. Gene-based analysis revealed a high enrichment
for olfactory receptor genes in this region. Phenome-wide
association studies demonstrated associations between
genetic variants related to olfactory dysfunction and blood
cell counts, kidney function, skeletal muscle mass,
cholesterol levels and cardiovascular disease. Using
individual-level data, we also confirmed and quantified the
strength of these associations on a phenotypic level.
Moreover, employing two-sample Mendelian Randomization
analyses, we found evidence for causal associations between
olfactory dysfunction and these phenotypes.Our findings
provide novel insights into the genetic architecture of the
sense of smell and highlight its importance for many aspects
of human health. Moreover, these findings could facilitate
the identification and monitoring of individuals at
increased risk of olfactory dysfunction and associated
diseases.},
keywords = {Humans / Genome-Wide Association Study / Olfaction
Disorders: genetics / Polymorphism, Single Nucleotide / Male
/ Female / Genetic Predisposition to Disease / Receptors,
Odorant: genetics / Middle Aged / White People: genetics /
Aged / White / Anthropometric (Other) / Biochemical (Other)
/ Gene-mapping (Other) / Genome-wide association
meta-analysis (Other) / Odor identification test (Other) /
Olfactory dysfunction (Other) / PheWAS (Other) / Sense of
smell (Other) / Two-sample MR (Other) / Receptors, Odorant
(NLM Chemicals)},
cin = {AG Aziz / AG Breteler},
ddc = {610},
cid = {I:(DE-2719)5000071 / I:(DE-2719)1012001},
pnm = {354 - Disease Prevention and Healthy Aging (POF4-354) /
TRANSIT-ND - Tandem Repeats Associated with Neurogenomic
Somatic Instability and Neurodegeneration (101041677)},
pid = {G:(DE-HGF)POF4-354 / G:(EU-Grant)101041677},
experiment = {EXP:(DE-2719)Rhineland Study-20190321},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40963118},
doi = {10.1186/s12863-025-01360-z},
url = {https://pub.dzne.de/record/281276},
}