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@ARTICLE{Klier:281359,
author = {Klier, Kristin and Mehrjerd, Ameneh and Fässler, Daniel
and Franck, Maximilien and Weihs, Antoine and Budde, Kathrin
and Bahls, Martin and Frost, Fabian and Henning, Ann-Kristin
and Heinken, Almut and Völzke, Henry and Dörr, Marcus and
Nauck, Matthias and Grabe, Hans Jörgen and Friedrich, Nele
and Hertel, Johannes},
title = {{I}ntegrating population-based metabolomics with
computational microbiome modelling identifies methanol as a
urinary biomarker for protective diet-microbiome-host
interactions.},
journal = {Food $\&$ function},
volume = {16},
number = {18},
issn = {2042-6496},
address = {Cambridge},
publisher = {RSC},
reportid = {DZNE-2025-01106},
pages = {7067 - 7081},
year = {2025},
abstract = {Background: Diet-microbiome interactions are core to human
health, in particular through bacterial fibre degradation
pathways. However, biomarkers reflective of these
interactions are not well described. Methods: Using the
population-based SHIP-START-0 cohort (n = 4017), we combined
metabolome-wide screenings with elastic net machine learning
models on 33 food items captured using a food frequency
questionnaire (FFQ) and 43 targeted urine nuclear magnetic
resonance (NMR) metabolites, identifying methanol as a
marker of plant-derived food items. We utilised the
independent SHIP-START-0 cohort for the replication of
food-metabolite associations. Moreover, constraint-based
microbiome community modelling using the Human Microbiome
data (n = 149) was performed to predict and analyse the
contribution of the microbiome to the human methanol pools
through bacterial fibre degradation. Finally, we employed
prospective survival analysis in the SHIP-START-0 cohort,
testing urinary methanol on its predictive value for
mortality. Results: Among 21 metabolites associated with 17
dietary FFQ variables after correction for multiple testing,
urinary methanol emerged as the top hit for a range of
plant-derived food items. In line with this,
constraint-based community modelling demonstrated that gut
microbiomes can produce methanol via pectin degradation with
the genera Bacteroides $(68.9\%)$ and Faecalibacterium
$(20.6\%)$ being primarily responsible. Moreover, microbial
methanol production capacity was a marker of high microbiome
diversity. Finally, prospective survival analysis in
SHIP-START-0 revealed that higher urinary methanol is
associated with lower all-cause mortality in fully adjusted
Cox regressions. Conclusion: Integrating population-based
metabolomics and computational microbiome modelling
identified urinary methanol as a promising biomarker for
protective diet-microbiome interactions linked to microbial
pectin degradation.},
keywords = {Humans / Biomarkers: urine / Methanol: urine / Methanol:
metabolism / Metabolomics / Male / Gastrointestinal
Microbiome: physiology / Female / Middle Aged / Diet / Adult
/ Host Microbial Interactions / Aged / Biomarkers (NLM
Chemicals) / Methanol (NLM Chemicals)},
cin = {AG Hoffmann / AG Grabe},
ddc = {610},
cid = {I:(DE-2719)1510600 / I:(DE-2719)5000001},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40856313},
doi = {10.1039/D5FO00761E},
url = {https://pub.dzne.de/record/281359},
}