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@ARTICLE{Methorst:281795,
author = {Methorst, Jeroen and Verwei, Nino and Hoffmann, Christian
and Chodnicki, Paweł and Sansevrino, Roberto and Pyne,
Partha and Wang, Han and van Hilten, Niek and Aschmann,
Dennis and Kros, Alexander and Andreas, Loren and Czub,
Jacek and Milovanovic, Dragomir and Risselada, Herre Jelger},
title = {{P}hysics-based evolution of transmembrane helices reveals
mechanisms of cholesterol attraction.},
journal = {Nature Communications},
volume = {16},
number = {1},
issn = {2041-1723},
address = {[London]},
publisher = {Springer Nature},
reportid = {DZNE-2025-01185},
pages = {9275},
year = {2025},
abstract = {The existence of linear cholesterol-recognition motifs in
transmembrane domains has long been debated. Evolutionary
molecular dynamics (Evo-MD) simulations-genetic algorithms
guided by (coarse-grained) molecular force-fields-reveal
that thermodynamic optimal cholesterol attraction in
isolated alpha-helical transmembrane domains occurs when
multiple consecutive lysine/arginine residues flank a short
hydrophobic segment. These findings are supported by
atomistic simulations and solid-state NMR experiments. Our
analyses illustrate that linear motifs in transmembrane
domains exhibit weak binding affinity for cholesterol,
characterized by sub-microsecond residence times,
challenging the predictive value of linear CRAC/CARC motifs
for cholesterol binding. Membrane protein database analyses
suggest even weaker affinity for native linear motifs,
whereas live cell assays demonstrate that optimizing
cholesterol binding restricts transmembrane domains to the
endoplasmic reticulum post-translationally. In summary,
these findings contribute to our understanding of
cholesterol-protein interactions and offer insight into the
mechanisms of protein-mediated cholesterol regulation within
membranes.},
keywords = {Cholesterol: metabolism / Cholesterol: chemistry /
Molecular Dynamics Simulation / Membrane Proteins: chemistry
/ Membrane Proteins: metabolism / Humans / Thermodynamics /
Cell Membrane: metabolism / Cell Membrane: chemistry /
Protein Binding / Endoplasmic Reticulum: metabolism / Amino
Acid Motifs / Hydrophobic and Hydrophilic Interactions /
Cholesterol (NLM Chemicals) / Membrane Proteins (NLM
Chemicals)},
cin = {AG Milovanovic (Berlin)},
ddc = {500},
cid = {I:(DE-2719)1813002},
pnm = {351 - Brain Function (POF4-351)},
pid = {G:(DE-HGF)POF4-351},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41115860},
doi = {10.1038/s41467-025-63769-5},
url = {https://pub.dzne.de/record/281795},
}