TY - JOUR
AU - Tschirner, Sarah K
AU - Schmidt, Andree
AU - Ito, Mana
AU - Hyakkoku, Kana
AU - Yoshimura, Akimasa
AU - Müller, Stephan A
AU - Horiguchi, Naotaka
AU - Lichtenthaler, Stefan F
TI - Elenbecestat and Compound 89 Potently Inhibit BACE1 but Not BACE2 When Subchronically Dosed in Non-Human Primates.
JO - Proteomics
VL - 26
IS - 1
SN - 1615-9853
CY - Weinheim
PB - Wiley VCH
M1 - DZNE-2026-00070
SP - 100 - 107
PY - 2026
AB - The β-secretase BACE1 (β-site amyloid precursor (APP) cleaving enzyme 1) is a major drug target for Alzheimer's disease (AD), as it catalyzes the first step in amyloid β (Aβ) generation, but has additional substrates and functions, in particular in the brain. Several advanced clinical trials with BACE1 inhibitors were stopped because of an adverse event, a mild cognitive worsening. The underlying mechanism is not yet known but may result from co-inhibition of the BACE1-homolog BACE2. While a cerebrospinal fluid (CSF) biomarker for measuring BACE2 activity is not yet established, VCAM-1 has been suggested as such a biomarker, but has not yet been tested upon prolonged dosing in vivo. Using CSF pharmacoproteomics and a subchronic dosing paradigm in non-human primates, we demonstrate that compound 89, a BACE inhibitor not yet tested in humans, and the clinically tested drug elenbecestat inhibit BACE1 in vivo, with little or no effect on BACE2, as seen with a reduction of substrates of BACE1, but not of the BACE2 substrate VCAM-1. As a control, verubecestat, which inhibits both BACE2 and BACE1, reduced CSF abundance of BACE1 substrates as well as of VCAM-1. This study demonstrates the suitability of VCAM-1 as a pharmacodynamic biomarker for measuring BACE2 target engagement in CSF.
KW - Amyloid Precursor Protein Secretases: antagonists & inhibitors
KW - Amyloid Precursor Protein Secretases: cerebrospinal fluid
KW - Animals
KW - Aspartic Acid Endopeptidases: antagonists & inhibitors
KW - Aspartic Acid Endopeptidases: cerebrospinal fluid
KW - Male
KW - Humans
KW - Biomarkers: cerebrospinal fluid
KW - Vascular Cell Adhesion Molecule-1: cerebrospinal fluid
KW - Macaca fascicularis
KW - Amyloid Precursor Protein Secretases (NLM Chemicals)
KW - Aspartic Acid Endopeptidases (NLM Chemicals)
KW - BACE1 protein, human (NLM Chemicals)
KW - Biomarkers (NLM Chemicals)
KW - Vascular Cell Adhesion Molecule-1 (NLM Chemicals)
LB - PUB:(DE-HGF)16
C6 - pmid:41310967
C2 - pmc:PMC12809003
DO - DOI:10.1002/pmic.70082
UR - https://pub.dzne.de/record/284027
ER -