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@ARTICLE{Antons:284041,
author = {Antons, Melissa J and Kloiber-Langhorst, Sandra and
Hirner-Eppeneder, Heidrun and Schaefer, Rebecca and Stueckl,
Jennifer and Palumbo, Giovanna and Oos, Rosel and Herr,
Felix L and Lindner, Simon and Ziegler, Sibylle and Brendel,
Matthias and Ricke, Jens and Werner, Rudolf A and Heimer,
Maurice M and Cyran, Clemens C},
title = {[18{F}]{FDG}-{PET}/{CT} {I}maging for {R}esponse
{C}haracterisation of {E}xperimental {M}elanomas to
{A}nti-{PD}-{L}1/{A}nti-{CTLA}-4 {I}mmunotherapy.},
journal = {Molecular imaging $\&$ biology},
volume = {27},
number = {6},
issn = {1536-1632},
address = {Cham},
publisher = {Springer Nature Switzerland},
reportid = {DZNE-2026-00076},
pages = {1006 - 1014},
year = {2025},
abstract = {Immune checkpoint inhibition has shown promising results in
malignant melanoma, but not all patients respond equally
well, necessitating early, accurate monitoring of
immunotherapy response. [18F]FDG-PET/CT aids in
characterising therapy response beyond morphology, but
validated imaging biomarkers for immunotherapy response
remain scarce. This study investigated three-time point
[18F]FDG-PET/CT to monitor combined anti-PD-L1/anti-CTLA-4
immunotherapy in murine melanoma allografts and compared
quantitative in vivo imaging biomarkers with ex vivo
biomarkers from multiparametric immunohistochemistry at each
time point.Melanoma cells (B16-F10) were injected
subcutaneously into C57BL/6 mice (n = 40). Seven days
post-inoculation, baseline [18F]FDG-PET/CT was conducted.
Animals were randomized into two groups; the therapy group
received 5 i.p.-injections of anti-PD-L1/anti-CTLA-4 (20
µg/kg) on days 7, 9, 11, 13 and 15 after tumor cell
inoculation. The control group received sham treatment.
PET/CT was performed at baseline (day 7 post inoculation),
follow-up 1(day 13; FU-1) and follow-up 2 (day 19; FU-2).
Tumor allografts were harvested at each time point for
immunohistochemistry (CD8, Ki-67, TUNEL) to validate imaging
parameters (MTV, SUVmax).At FU-1, the therapy group
exhibited significantly lower MTV than the control group (p
= 0.004). At FU-2, MTV and SUVmax were significantly lower
(MTV: p = 0.008; SUVmax: p = 0.0003) compared to controls.
Ex vivo analysis revealed significant anti-tumor effects in
the therapy group, with higher apoptosis rates (FU-1: p =
0.012; FU-2: p = 0.001), more CD8-positive T-cells (FU-2: p
= 0.003) and lower tumor cell proliferation (FU-1: p =
0.012; FU-2: p = 0.012).Multi-time point [18F]FDG-PET/CT
allowed for early non-invasive monitoring of combined
anti-PD-L1/anti-CTLA-4 immunotherapy in experimental
melanomas, validated by multiparametric immunohistochemistry
with significant pro-immunogenic, pro-apoptotic and
anti-proliferative effects.},
keywords = {Animals / Positron Emission Tomography Computed Tomography
/ Fluorodeoxyglucose F18: chemistry / Immunotherapy / CTLA-4
Antigen: antagonists $\&$ inhibitors / CTLA-4 Antigen:
immunology / Mice, Inbred C57BL / B7-H1 Antigen: antagonists
$\&$ inhibitors / B7-H1 Antigen: immunology / Cell Line,
Tumor / Melanoma, Experimental: diagnostic imaging /
Melanoma, Experimental: therapy / Melanoma, Experimental:
pathology / Melanoma, Experimental: immunology / Mice /
Female / Immune Checkpoint Inhibitors: therapeutic use /
Immune Checkpoint Inhibitors: pharmacology /
Immunohistochemistry (Other) / Immunotherapy (Other) /
Melanoma (Other) / [18F]FDG-PET/CT (Other) /
Fluorodeoxyglucose F18 (NLM Chemicals) / CTLA-4 Antigen (NLM
Chemicals) / B7-H1 Antigen (NLM Chemicals) / Immune
Checkpoint Inhibitors (NLM Chemicals)},
cin = {AG Haass},
ddc = {570},
cid = {I:(DE-2719)1110007},
pnm = {352 - Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41102571},
doi = {10.1007/s11307-025-02056-7},
url = {https://pub.dzne.de/record/284041},
}