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@ARTICLE{Kennedy:284043,
author = {Kennedy, James T and Wisch, Julie K and Boerwinkle, Anna H
and Millar, Peter R and McKay, Nicole S and Brickman, Adam M
and Chhatwal, Jasmeer P and Mendez, Patricio Chrem and
Christian, Bradley T and Cohen, Anne and Cruchaga, Carlos
and Daniels, Alisha and Flores, Shaney and Handen, Benjamin
L and Hartley, Sigan L and Head, Elizabeth and Ibanez, Laura
and Krisnsky-McHale, Sharon J and la Fougere, Christian and
Lai, Florence and Laymon, Charles M and Lee, Joseph H and
Lee, Jae-Hong and Levin, Johannes and Llibre-Guerra, Jorge
and Aguillon, David F and Lott, Ira T and Mapstone, Mark and
McDade, Eric and Morris, John and O'Bryant, Sid E and Price,
Julie C and Rafii, Michael S and Roh, Jee Hoon and Rosas, H
Diana and Schupf, Nicole and Supnet-Bell, Charlene and
Xiong, Chengjie and Zaman, Shahid and Benzinger, Tammie L S
and Gordon, Brian A and Ances, Beau M and Alzheimer's Brain
Consortium - Down Syndrome},
collaboration = {Network, the Dominantly Inherited Alzheimer's},
title = {{B}rain volume trajectories in {D}own syndrome and
autosomal dominant {A}lzheimer's disease.},
journal = {Alzheimer's and dementia},
volume = {22},
number = {1},
issn = {1552-5260},
address = {Hoboken, NJ},
publisher = {Wiley},
reportid = {DZNE-2026-00078},
pages = {e71103},
year = {2026},
abstract = {It is unknown if neurodegeneration trajectories differ
between Down syndrome (DS) and autosomal dominant
Alzheimer's disease (ADAD), both of which are genetic forms
of Alzheimer's disease (AD).We compared brain volumes in DS,
ADAD, and unaffected family members serving as controls.
Participants underwent magnetic resonance imaging (MRI) and
amyloid positron emission tomography (PET), deriving
volumetric and amyloid burden, respectively. Nonlinear
associations between regional volumes and estimated years to
clinical symptom onset (EYO) were evaluated using
generalized additive mixed-models.Longitudinal data from 267
controls, 341 participants with DS, and 358 participants
with ADAD were included, totaling 1908 scans. DS volumes
were lower than ADAD and controls initially and dropped
linearly. ADAD had similar volumes to controls until
diverging, beginning at EYO -7. Amyloid was negatively
associated with volume, with similar slopes in DS and
ADAD.ADAD and DS demonstrate distinct patterns of brain
volume decline prior to symptom onset despite being
similarly affected by amyloid.},
keywords = {Humans / Down Syndrome: pathology / Down Syndrome:
diagnostic imaging / Alzheimer Disease: pathology /
Alzheimer Disease: diagnostic imaging / Alzheimer Disease:
genetics / Male / Female / Brain: pathology / Brain:
diagnostic imaging / Magnetic Resonance Imaging /
Positron-Emission Tomography / Middle Aged / Longitudinal
Studies / Organ Size / Adult / Disease Progression / Aged /
MRI (Other) / amyloid (Other) / volume (Other)},
cin = {Clinical Research (Munich) / AG Levin},
ddc = {610},
cid = {I:(DE-2719)1111015 / I:(DE-2719)1111016},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
experiment = {EXP:(DE-2719)DIAN-20090101},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41549404},
doi = {10.1002/alz.71103},
url = {https://pub.dzne.de/record/284043},
}