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@ARTICLE{Runge:284044,
author = {Runge, Kimon and Feige, Bernd and Schiele, Miriam A and von
Zedtwitz, Katharina and Maier, Alexander and Stöcker, Nils
and Dressle, Raphael J and Baldermann, Juan C and Maier,
Simon J and Nickel, Kathrin and Prüss, Harald and Coenen,
Volker Arnd and Voderholzer, Ulrich and Domschke, Katharina
and Tebartz van Elst, Ludger and Endres, Dominique},
title = {{S}econdary obsessive-compulsive syndromes: a systematic
literature review resulting in 228 suspected cases.},
journal = {Molecular psychiatry},
volume = {31},
number = {2},
issn = {1359-4184},
address = {[London]},
publisher = {Springer Nature},
reportid = {DZNE-2026-00079},
pages = {1121 - 1131},
year = {2026},
abstract = {Secondary forms of obsessive-compulsive disorder (OCD) have
clear underlying organic causes and are recognized as
distinct nosological entities in the latest international
classification systems. This study aims to provide a
systematic overview of published cases of suspected
secondary obsessive-compulsive syndromes. A systematic
literature search of PubMed, Embase, Web of Science, and
PsycINFO was conducted oriented on PRISMA criteria. Cases
from case studies/series of patients with suspected
secondary obsessive-compulsive syndromes and/or secondary
obsessive-compulsive symptoms were included. Cases of
obsessive-compulsive symptoms due to pediatric acute-onset
neuropsychiatric syndrome (PANS) and pediatric autoimmune
neuropsychiatric disorders associated with streptococcal
infections (PANDAS) were excluded. Overall, 228 cases of
suspected secondary obsessive-compulsive syndromes were
identified from 189 publications. Causal factors included
brain lesions $(25.4\%),$ genetic syndromes $(24.1\%),$ head
trauma $(12.3\%),$ autoimmune-inflammatory processes
$(11.8\%),$ tumors $(8.3\%),$ neurodegeneration $(7.5\%),$
seizures $(4.8\%),$ pathogens $(3.9\%),$ metabolic processes
$(1.3\%),$ or other reasons $(0.4\%).$ The age of the
affected patients varied considerably (mean 37.3 ± 21.2
years, range 4-94 years, n = 226). Diagnostic abnormalities
were identified through brain imaging (magnetic resonance
imaging/computer tomography) in $66.2\%$ of the sample and
via blood analysis in $23.9\%.$ In cases reporting the
regions of the brain involvement, frontal lobe $(34.3\%)$
and the basal ganglia $(26.5\%)$ were mostly affected. The
findings highlight a variety of suspected causes of
secondary obsessive-compulsive syndromes, most frequently
brain lesions, genetic syndromes, head trauma, and
autoimmune-inflammatory processes. Identifying secondary
obsessive-compulsive symptoms informed personalized
therapies in a subgroup of published cases.},
keywords = {Adolescent / Adult / Child / Female / Humans / Male /
Middle Aged / Autoimmune Diseases: complications / Brain /
Obsessive-Compulsive Disorder: etiology /
Obsessive-Compulsive Disorder: diagnosis / Streptococcal
Infections},
cin = {AG Prüß},
ddc = {610},
cid = {I:(DE-2719)1810003},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41436581},
pmc = {pmc:PMC12815680},
doi = {10.1038/s41380-025-03395-1},
url = {https://pub.dzne.de/record/284044},
}