000285003 001__ 285003 000285003 005__ 20260202112928.0 000285003 0247_ $$2doi$$a10.1007/s00259-025-07579-3 000285003 0247_ $$2pmid$$apmid:41114734 000285003 0247_ $$2ISSN$$a1619-7070 000285003 0247_ $$2ISSN$$a1619-7089 000285003 037__ $$aDZNE-2026-00137 000285003 041__ $$aEnglish 000285003 082__ $$a610 000285003 1001_ $$aCrook, Harry$$b0 000285003 245__ $$aComparing and combining TSPO-PET tracers in tauopathies. 000285003 260__ $$aHeidelberg [u.a.]$$bSpringer-Verl.$$c2026 000285003 3367_ $$2DRIVER$$aarticle 000285003 3367_ $$2DataCite$$aOutput Types/Journal article 000285003 3367_ $$0PUB:(DE-HGF)16$$2PUB:(DE-HGF)$$aJournal Article$$bjournal$$mjournal$$s1770027558_15532 000285003 3367_ $$2BibTeX$$aARTICLE 000285003 3367_ $$2ORCID$$aJOURNAL_ARTICLE 000285003 3367_ $$00$$2EndNote$$aJournal Article 000285003 520__ $$aNeuroinflammation is a key pathological driver in neurodegenerative diseases, including Alzheimer's disease (AD) and Progressive Supranuclear Palsy (PSP). Positron emission tomography (PET) with tracers targeting the translocator protein (TSPO) enables the in vivo quantification of microgliosis. TSPO tracers have shown similar disease-specific patterns across cohorts. However, direct quantitative comparisons between commonly used TSPO-PET tracers in tauopathies have not been performed. Here, we apply a TSPO-PET standardization pipeline across clinically matched AD cohorts and PSP cohorts, to quantify, compare and combine multi-centre TSPO-PET data.Patients with PSP were scanned with either [11C]PK11195 or [18F]GE-180 at one of two centres, while patients with AD and control participants were scanned with either [11C]PK11195, [18F]GE-180 or [11C]PBR28 at one of three centres. A standardised pre-processing pipeline was implemented and participant standardised uptake volume ratio (SUVR) values were z-scored using tracer-specific control participant values. In a data-driven approach, dissimilarity analyses were employed to assess differences between tracers across clinically matched cohorts.In PSP, dissimilarity analysis suggested that [11C]PK11195 and [18F]GE-180 binding patterns were comparable following standardisation. In AD, comparability across tracers was less robust, with [11C]PK11195 and [18F]GE-180 being most comparable, followed by [18F]GE-180 vs. [11C]PBR28, then by [11C]PK11195 vs. [11C]PBR28.The pipeline was effective at harmonising TSPO-PET tracers and standardising the regional quantification of neuroinflammation in clinically matched cohorts of PSP, while the standardisation pipeline results were less robust across AD cohorts. 000285003 536__ $$0G:(DE-HGF)POF4-353$$a353 - Clinical and Health Care Research (POF4-353)$$cPOF4-353$$fPOF IV$$x0 000285003 536__ $$0G:(DE-HGF)POF4-352$$a352 - Disease Mechanisms (POF4-352)$$cPOF4-352$$fPOF IV$$x1 000285003 588__ $$aDataset connected to CrossRef, PubMed, , Journals: pub.dzne.de 000285003 650_7 $$2Other$$aAlzheimer’s disease. 000285003 650_7 $$2Other$$aNeuroinflammation 000285003 650_7 $$2Other$$aPositron emission tomography 000285003 650_7 $$2Other$$aProgressive supranuclear palsy 000285003 650_7 $$2Other$$aTranslocator protein 000285003 650_7 $$2NLM Chemicals$$aReceptors, GABA 000285003 650_7 $$2NLM Chemicals$$aTSPO protein, human 000285003 650_7 $$2NLM Chemicals$$aRadioactive Tracers 000285003 650_7 $$0YNF83VN1RL$$2NLM Chemicals$$aPK 11195 000285003 650_7 $$2NLM Chemicals$$aGE-180 000285003 650_7 $$2NLM Chemicals$$aRadiopharmaceuticals 000285003 650_7 $$2NLM Chemicals$$aCarbazoles 000285003 650_7 $$2NLM Chemicals$$aIsoquinolines 000285003 650_2 $$2MeSH$$aHumans 000285003 650_2 $$2MeSH$$aPositron-Emission Tomography: methods 000285003 650_2 $$2MeSH$$aPositron-Emission Tomography: standards 000285003 650_2 $$2MeSH$$aReceptors, GABA: metabolism 000285003 650_2 $$2MeSH$$aMale 000285003 650_2 $$2MeSH$$aFemale 000285003 650_2 $$2MeSH$$aAged 000285003 650_2 $$2MeSH$$aMiddle Aged 000285003 650_2 $$2MeSH$$aRadioactive Tracers 000285003 650_2 $$2MeSH$$aTauopathies: diagnostic imaging 000285003 650_2 $$2MeSH$$aTauopathies: metabolism 000285003 650_2 $$2MeSH$$aAlzheimer Disease: diagnostic imaging 000285003 650_2 $$2MeSH$$aAlzheimer Disease: metabolism 000285003 650_2 $$2MeSH$$aRadiopharmaceuticals 000285003 650_2 $$2MeSH$$aCarbazoles 000285003 650_2 $$2MeSH$$aIsoquinolines 000285003 7001_ $$aFranzmeier, Nicolai$$b1 000285003 7001_ $$aRahmouni, Nesrine$$b2 000285003 7001_ $$0P:(DE-2719)9001652$$aGnoerich, Johannes$$b3$$udzne 000285003 7001_ $$aFryer, Tim D$$b4 000285003 7001_ $$aHong, Young T$$b5 000285003 7001_ $$aRoemer-Cassiano, Sebastian N$$b6 000285003 7001_ $$0P:(DE-2719)9000852$$aPalleis, Carla$$b7$$udzne 000285003 7001_ $$aStrauss, Alexandra$$b8 000285003 7001_ $$aJones, P Simon$$b9 000285003 7001_ $$aAigbirhio, Franklin I$$b10 000285003 7001_ $$aHopewell, Robert$$b11 000285003 7001_ $$0P:(DE-2719)9001808$$aRauchmann, Boris Stephan$$b12$$udzne 000285003 7001_ $$aMassarweh, Gassan$$b13 000285003 7001_ $$0P:(DE-2719)2812234$$aPerneczky, Robert$$b14$$udzne 000285003 7001_ $$0P:(DE-2719)2811659$$aLevin, Johannes$$b15$$udzne 000285003 7001_ $$0P:(DE-2719)2811373$$aHöglinger, Günter U$$b16$$udzne 000285003 7001_ $$aRowe, James B$$b17 000285003 7001_ $$aO'Brien, John T$$b18 000285003 7001_ $$aRosa-Neto, Pedro$$b19 000285003 7001_ $$0P:(DE-2719)9001539$$aBrendel, Matthias$$b20$$udzne 000285003 7001_ $$00000-0001-8923-9656$$aMalpetti, Maura$$b21 000285003 773__ $$0PERI:(DE-600)2098375-X$$a10.1007/s00259-025-07579-3$$gVol. 53, no. 3, p. 2083 - 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