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@ARTICLE{Tong:285207,
author = {Tong, Benjamin Chun-Kit and Gubinelli, Francesco and
Burbulla, Lena F and Harbauer, Angelika B},
title = {{M}itochondrial specialization and signaling shape neuronal
function.},
journal = {Trends in neurosciences},
volume = {49},
number = {2},
issn = {0378-5912},
address = {Amsterdam [u.a.]},
publisher = {Elsevier Science},
reportid = {DZNE-2026-00186},
pages = {141 - 154},
year = {2026},
abstract = {Neurons are specialized cells designed to process
information and transmit it, often across long distances. In
many neurons, the axonal volume far exceeds the
somato-dendritic volume, creating a need for long-range
transport and local polarization mechanisms. In addition,
action potential firing and restoration of ionic gradients,
as well as dynamic changes in synaptic plasticity, further
increase the energetic demands of neurons. In this review,
we highlight the roles mitochondria play in vertebrate
neuronal biology and how mitochondrial functionality is
tuned to support the unique demands of neurons. We cover the
influence of mitochondrial positioning, ATP generation and
Ca2+ buffering on neuronal function, and explore the role of
mitochondria in neurotransmitter metabolism and local
protein translation.},
keywords = {Mitochondria: physiology / Mitochondria: metabolism /
Animals / Neurons: physiology / Neurons: metabolism / Humans
/ Signal Transduction: physiology / Neuronal Plasticity:
physiology / Calcium: metabolism / Ca(2+) signaling (Other)
/ local translation (Other) / neuronal cell biology (Other)
/ neurotransmitter metabolism (Other) / respiration (Other)
/ transport (Other) / Calcium (NLM Chemicals)},
cin = {AG Burbulla},
ddc = {610},
cid = {I:(DE-2719)5000074},
pnm = {352 - Disease Mechanisms (POF4-352)},
pid = {G:(DE-HGF)POF4-352},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41638946},
doi = {10.1016/j.tins.2025.12.006},
url = {https://pub.dzne.de/record/285207},
}