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@ARTICLE{Maass:285457,
author = {Maass, Fabian and Thomas, Carolina and Kurvits, Lille and
Hermann, Peter and Schmitz, Matthias and Bähr, Mathias and
Stadelmann, Christine and Zechel, Sabrina and Canaslan,
Sezgi and Zerr, Inga},
title = {{A}lterations in cerebrospinal fluid levels of myelin- and
oligodendrocyte-related proteins in sporadic
{C}reutzfeldt–{J}akob disease},
journal = {Acta Neuropathologica Communications},
volume = {14},
number = {1},
issn = {2051-5960},
address = {London},
publisher = {Biomed Central},
reportid = {DZNE-2026-00239},
pages = {51},
year = {2026},
abstract = {Recent evidence suggests glial dysfunction, particularly
involving oligodendrocytes and myelin, as an important part
of the primary disease mechanism of sporadic
Creutzfeldt–Jakob disease. To the best of our knowledge,
cerebrospinal fluid (CSF) biomarkers reflecting
oligodendrocyte or myelin damage have not yet been
systematically investigated in this context. CSF samples
from patients with sporadic Creutzfeldt–Jakob disease
(CJD), Alzheimer's disease (AD) and non-neurodegenerative
controls (n = 18, respectively) were included in the study,
and levels of myelin basic protein (MBP), neural/glial
antigen 2 (NG2) and cyclic nucleotide 3′-phosphodiesterase
(CNPase) were quantified. Additionally, p25-positive cells
were quantified in autopsy tissue from the frontal and
parietal cortical regions of five patients with CJD and four
controls. In the primary cohort, MBP quantification revealed
significantly higher CSF levels in CJD compared to AD (p =
0.004) and controls (p = 0.0001) and this difference
remained significant after adjustment for age and
neurofilament light chain (NfL), indicating that MBP
elevations cannot be explained solely by neuroaxonal
degeneration. Significant differences were also found for
CNPase in CJD compared to AD (p = 0.0011) and controls (p =
0.0004). There were no differences in NG2 levels (p > 0.05)
among the groups. However, no significant differences were
observed in the total number of p25-positive mature
oligodendrocytes between CJD and controls in cortical brain
tissue (p > 0.05). Additionally, MBP and NfL were quantified
in a second cohort of 28 CJD and 20 biomarker-defined AD
patients. In this cohort, characterized by more advanced AD
pathology, MBP strongly covaried with NfL and no longer
differed between diagnostic groups, suggesting a greater
influence of neuroaxonal injury on MBP levels in advanced
disease stages. In conclusion, in CJD, MBP elevations appear
to be only partly related to neuroaxonal degeneration and
may reflect disease-related myelin involvement.The online
version contains supplementary material available at
10.1186/s40478-026-02247-5.},
keywords = {Biomarker (Other) / CNPase (Other) / Cerebrospinal fluid
(Other) / Creutzfeldt–Jakob disease (Other) / Myelin basic
protein (Other) / NG2 (Other)},
cin = {AG Zerr / Clinical Dementia Research (Göttingen)},
ddc = {610},
cid = {I:(DE-2719)1440011-1 / I:(DE-2719)1440015},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
typ = {PUB:(DE-HGF)16},
doi = {10.1186/s40478-026-02247-5},
url = {https://pub.dzne.de/record/285457},
}