TY - JOUR
AU - Ruf, Wolfgang P
AU - Kühlwein, Julia
AU - Meier, Laura
AU - Tripke, Sarah
AU - LeeBae, Jaehyun
AU - Sadri-Vakili, Ghazaleh
AU - Yilmazer-Hanke, Deniz
AU - Petri, Susanne
AU - Thal, Dietmar R
AU - Grozdanov, Veselin
AU - Danzer, Karin M
TI - Multi-modal dissection of cell-type specific TDP-43 pathology in the motor cortex.
JO - Nature Communications
VL - 17
IS - 1
SN - 2041-1723
CY - [London]
PB - Springer Nature
M1 - DZNE-2026-00277
SP - 2406
PY - 2026
AB - Cytoplasmic TDP-43 pathology is a pathological sign of ALS/ALS-FTD and a converging disease event across different genotypes, phenotypes and CNS areas. To understand this process and target it therapeutically, we need to define which cell types are affected and which cell-type specific effects make them particularly vulnerable. We coupled flow-cytometry nuclear sorting and sequencing with single-nucleus multi-omic ATAC-seq and RNA-seq and spatial transcriptomics to define the transcriptional cell type of affected neurons in the post-mortem ALS/ALS-FTD motor cortex (30 ALS, 20 ALS-FTD </td><td width="150">
AB - 32 control samples). Here, we show that mainly excitatory cortical neurons are affected by TDP-43 pathology and define the cell types that are affected the most: intratelencephalic L2-L3-LINC00507-FREM3, L3-L5-RORB-LNX2, L3-L5-RORB-ADGRL4 </td><td width="150">
AB - L6-THEMIS-LINC00343 neurons and extratelencephalic L5-FEZF2-NTNG1 neurons. Transcriptional aberrations by TDP-43 pathology, like cryptic exon inclusion, are cell-type specific and affect distinct gene sets in each cell type, highlighting the need to address TDP-43 pathology in a cell-type specific manner.
KW - Motor Cortex: pathology
KW - Motor Cortex: metabolism
KW - Humans
KW - DNA-Binding Proteins: metabolism
KW - DNA-Binding Proteins: genetics
KW - Amyotrophic Lateral Sclerosis: pathology
KW - Amyotrophic Lateral Sclerosis: genetics
KW - Amyotrophic Lateral Sclerosis: metabolism
KW - Male
KW - Neurons: metabolism
KW - Neurons: pathology
KW - Female
KW - Frontotemporal Dementia: pathology
KW - Frontotemporal Dementia: genetics
KW - Frontotemporal Dementia: metabolism
KW - Aged
KW - Middle Aged
KW - Transcriptome
KW - DNA-Binding Proteins (NLM Chemicals)
KW - TARDBP protein, human (NLM Chemicals)
LB - PUB:(DE-HGF)16
C6 - pmid:41803120
DO - DOI:10.1038/s41467-026-69944-6
UR - https://pub.dzne.de/record/285638
ER -