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@ARTICLE{Abrous:285922,
author = {Abrous, Djoher Nora and Blin, Nicolas and Boraxbekk,
Carl-Johan and Catheline, Gwenaelle and Fitzsimons, Carlos P
and Hilscher, Markus and Lemoine, Maël and Lopes, Luísa V
and Maass, Anne and Nilsson, Mats and Nyberg, Lars and
Rassmussen, Lene Juel and Remondes, Miguel and Sauvage,
Magdalena and Schreiber, Stefanie and Wolbers, Thomas},
title = {{H}allmarks of healthy cognitive aging: inter-individual
differences in aging trajectories.},
journal = {Ageing research reviews},
volume = {Advance online publication},
issn = {1568-1637},
address = {Oxford [u.a.]},
publisher = {Elsevier},
reportid = {DZNE-2026-00368},
pages = {103102},
year = {2026},
abstract = {Cognitive aging is a highly heterogeneous process, with
some individuals preserving stable cognitive performance
across the lifespan while others exhibiting pronounced
decline. This marked interindividual variability indicates
that chronological age alone is a poor predictor of
cognitive health. Rather than reflecting uniform
degeneration, cognitive aging emerges from divergent
biological trajectories spanning molecular, cellular, and
network levels. In this review, we synthesize emerging
biological hallmarks of healthy cognitive aging, emphasizing
studies that characterize longitudinal cognitive
trajectories in humans or distinguish aged individuals who
retain learning capacity from those who do not. We focus on
the medial temporal lobe, a region critical for episodic
memory and spatial navigation, and examine how variability
in its integrity contributes to distinct cognitive outcomes.
Across species, convergent evidence suggests that cognitive
decline is more closely linked to alterations in network
regulation and synaptic plasticity than to overt neuronal
loss. We identify key mechanisms shaping individual
trajectories, including large-scale network organization,
excitation-inhibition balance, neuromodulatory tone, glial
and vascular regulation, adult hippocampal neurogenesis, and
cellular homeostasis. These processes operate within an
interconnected system in which disruptions in core
regulatory mechanisms can propagate across levels of
organization. Together, this synthesis supports a
system-level framework in which cognitive resilience depends
on the preservation of coordinated network dynamics. We
advocate for longitudinal, multidimensional approaches to
identify early shifts in regulatory balance and inform
strategies to maintain cognitive function across the
lifespan.},
subtyp = {Review Article},
keywords = {Cognitive Aging (Other) / Episodic Memory (Other) /
Hallmarks (Other) / inter-individual differences (Other) /
medial temporal lobe (Other) / spatial memory (Other)},
cin = {AG Wolbers / AG Maaß},
ddc = {610},
cid = {I:(DE-2719)1310002 / I:(DE-2719)1311001},
pnm = {353 - Clinical and Health Care Research (POF4-353)},
pid = {G:(DE-HGF)POF4-353},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41865894},
doi = {10.1016/j.arr.2026.103102},
url = {https://pub.dzne.de/record/285922},
}