%0 Journal Article
%A Pradhan, Ranjit
%A Sakib, M Sadman
%A Kaurani, Lalit
%A Krüger, Dennis M
%A Pena, Tonatiuh
%A Burkhardt, Susanne
%A Schütz, Anna-Lena
%A Kronenberg-Versteeg, Deborah
%A Delalle, Ivana
%A Sananbenesi, Farahnaz
%A Fischer, Andre
%T lncRNA Glelr modulates microglia inflammatory programs in association with PU.1.
%J Neurobiology of disease
%V 222
%@ 0969-9961
%C [Amsterdam]
%I Elsevier
%M DZNE-2026-00385
%P 107366
%D 2026
%X Long non-coding RNAs (lncRNAs) are emerging as key regulators of brain function, but their contribution to microglial aging and neurodegenerative disease remains largely unknown. Because only 1.5
%K RNA, Long Noncoding: metabolism
%K RNA, Long Noncoding: genetics
%K Microglia: metabolism
%K Animals
%K Humans
%K Mice
%K Trans-Activators: metabolism
%K Trans-Activators: genetics
%K Proto-Oncogene Proteins: metabolism
%K Proto-Oncogene Proteins: genetics
%K Inflammation: metabolism
%K Inflammation: genetics
%K Mice, Inbred C57BL
%K Cells, Cultured
%K Aging: metabolism
%K Astrocytes: metabolism
%K 3222401L13Rik/ENSG00000272070 (Other)
%K Alzheimer's disease (Other)
%K Long non-coding RNA (lncRNA) (Other)
%K Microglia (Other)
%K Neuroinflammation (Other)
%K Non-coding RNAome (Other)
%K PU.1 (SPI1) (Other)
%K RNA, Long Noncoding (NLM Chemicals)
%K Trans-Activators (NLM Chemicals)
%K Proto-Oncogene Proteins (NLM Chemicals)
%F PUB:(DE-HGF)16
%9 Journal Article
%$ pmid:41895620
%R 10.1016/j.nbd.2026.107366
%U https://pub.dzne.de/record/286089