TY  - JOUR
AU  - Pradhan, Ranjit
AU  - Sakib, M Sadman
AU  - Kaurani, Lalit
AU  - Krüger, Dennis M
AU  - Pena, Tonatiuh
AU  - Burkhardt, Susanne
AU  - Schütz, Anna-Lena
AU  - Kronenberg-Versteeg, Deborah
AU  - Delalle, Ivana
AU  - Sananbenesi, Farahnaz
AU  - Fischer, Andre
TI  - lncRNA Glelr modulates microglia inflammatory programs in association with PU.1.
JO  - Neurobiology of disease
VL  - 222
SN  - 0969-9961
CY  - [Amsterdam]
PB  - Elsevier
M1  - DZNE-2026-00385
SP  - 107366
PY  - 2026
AB  - Long non-coding RNAs (lncRNAs) are emerging as key regulators of brain function, but their contribution to microglial aging and neurodegenerative disease remains largely unknown. Because only 1.5
KW  - RNA, Long Noncoding: metabolism
KW  - RNA, Long Noncoding: genetics
KW  - Microglia: metabolism
KW  - Animals
KW  - Humans
KW  - Mice
KW  - Trans-Activators: metabolism
KW  - Trans-Activators: genetics
KW  - Proto-Oncogene Proteins: metabolism
KW  - Proto-Oncogene Proteins: genetics
KW  - Inflammation: metabolism
KW  - Inflammation: genetics
KW  - Mice, Inbred C57BL
KW  - Cells, Cultured
KW  - Aging: metabolism
KW  - Astrocytes: metabolism
KW  - 3222401L13Rik/ENSG00000272070 (Other)
KW  - Alzheimer's disease (Other)
KW  - Long non-coding RNA (lncRNA) (Other)
KW  - Microglia (Other)
KW  - Neuroinflammation (Other)
KW  - Non-coding RNAome (Other)
KW  - PU.1 (SPI1) (Other)
KW  - RNA, Long Noncoding (NLM Chemicals)
KW  - Trans-Activators (NLM Chemicals)
KW  - Proto-Oncogene Proteins (NLM Chemicals)
LB  - PUB:(DE-HGF)16
C6  - pmid:41895620
DO  - DOI:10.1016/j.nbd.2026.107366
UR  - https://pub.dzne.de/record/286089
ER  -