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000140884 0247_ $$2doi$$a10.1002/hipo.23078
000140884 0247_ $$2pmid$$apmid:30794330
000140884 0247_ $$2ISSN$$a1050-9631
000140884 0247_ $$2ISSN$$a1098-1063
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000140884 037__ $$aDZNE-2020-07206
000140884 041__ $$aEnglish
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000140884 1001_ $$0P:(DE-HGF)0$$aKnauer, Beate$$b0
000140884 245__ $$aSwitching between persistent firing and depolarization block in individual rat CA1 pyramidal neurons.
000140884 260__ $$aNew York, NY [u.a.]$$bWiley$$c2019
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000140884 520__ $$aThe hippocampal formation plays a role in mnemonic tasks and epileptic discharges in vivo. In vitro, these functions and malfunctions may relate to persistent firing (PF) and depolarization block (DB), respectively. Pyramidal neurons of the CA1 field have previously been reported to engage in either PF or DB during cholinergic stimulation. However, it is unknown whether these cells constitute disparate populations of neurons. Furthermore, it is unclear which cell-specific peculiarities may mediate their diverse response properties. However, it has not been shown whether individual CA1 pyramidal neurons can switch between PF and DB states. Here, we used whole cell patch clamp in the current clamp mode on in vitro CA1 pyramidal neurons from acutely sliced rat tissue to test various intrinsic properties which may provoke individual cells to switch between PF and DB. We found that individual cells could switch from PF to DB, in a cholinergic agonist concentration dependent manner and depending on the parameters of stimulation. We also demonstrate involvement of TRPC and potassium channels in this switching. Finally, we report that the probability for DB was more pronounced in the proximal than in the distal half of CA1. These findings offer a potential mechanism for the stronger spatial modulation in proximal, compared to distal CA1, as place field formation was shown to be affected by DB. Taken together, our results suggest that PF and DB are not mutually exclusive response properties of individual neurons. Rather, a cell's response mode depends on a variety of intrinsic properties, and modulation of these properties enables switching between PF and DB.
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000140884 542__ $$2Crossref$$i2019-02-22$$uhttp://doi.wiley.com/10.1002/tdm_license_1.1
000140884 542__ $$2Crossref$$i2019-02-22$$uhttp://onlinelibrary.wiley.com/termsAndConditions#vor
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000140884 650_2 $$2MeSH$$aAnimals
000140884 650_2 $$2MeSH$$aCA1 Region, Hippocampal: cytology
000140884 650_2 $$2MeSH$$aCA1 Region, Hippocampal: drug effects
000140884 650_2 $$2MeSH$$aCA1 Region, Hippocampal: physiology
000140884 650_2 $$2MeSH$$aCarbachol: pharmacology
000140884 650_2 $$2MeSH$$aDose-Response Relationship, Drug
000140884 650_2 $$2MeSH$$aElectrophysiological Phenomena: drug effects
000140884 650_2 $$2MeSH$$aElectrophysiological Phenomena: physiology
000140884 650_2 $$2MeSH$$aFemale
000140884 650_2 $$2MeSH$$aIn Vitro Techniques
000140884 650_2 $$2MeSH$$aMale
000140884 650_2 $$2MeSH$$aMembrane Potentials: drug effects
000140884 650_2 $$2MeSH$$aMuscarinic Agonists: pharmacology
000140884 650_2 $$2MeSH$$aPatch-Clamp Techniques
000140884 650_2 $$2MeSH$$aPotassium Channels: metabolism
000140884 650_2 $$2MeSH$$aPyramidal Cells: drug effects
000140884 650_2 $$2MeSH$$aPyramidal Cells: physiology
000140884 650_2 $$2MeSH$$aRats
000140884 650_2 $$2MeSH$$aRats, Long-Evans
000140884 650_2 $$2MeSH$$aTRPC Cation Channels: antagonists & inhibitors
000140884 650_2 $$2MeSH$$aTRPC Cation Channels: metabolism
000140884 7001_ $$0P:(DE-2719)2811873$$aYoshida, Motoharu$$b1$$eLast author$$udzne
000140884 77318 $$2Crossref$$3journal-article$$a10.1002/hipo.23078$$b : Wiley, 2019-02-22$$n9$$p817-835$$tHippocampus$$v29$$x1050-9631$$y2019
000140884 773__ $$0PERI:(DE-600)1498049-6$$a10.1002/hipo.23078$$gVol. 29, no. 9, p. 817 - 835$$n9$$p817-835$$q29:9<817 - 835$$tHippocampus$$v29$$x1050-9631$$y2019
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000140884 999C5 $$2Crossref$$oWitter Hippocampal microcircuits-A computational modeler's resource book 2010$$tHippocampal microcircuits-A computational modeler's resource book$$y2010
000140884 999C5 $$2Crossref$$9-- missing cx lookup --$$a10.1016/j.yjmcc.2004.11.020$$o10.1016/j.yjmcc.2004.11.020
000140884 999C5 $$2Crossref$$9-- missing cx lookup --$$a10.1523/JNEUROSCI.1042-09.2009$$o10.1523/JNEUROSCI.1042-09.2009
000140884 999C5 $$2Crossref$$9-- missing cx lookup --$$a10.3389/fncir.2012.00010$$o10.3389/fncir.2012.00010
000140884 999C5 $$2Crossref$$9-- missing cx lookup --$$a10.1152/jn.01333.2005$$o10.1152/jn.01333.2005
000140884 999C5 $$2Crossref$$oZawar 1999$$y1999
000140884 999C5 $$2Crossref$$9-- missing cx lookup --$$a10.1002/hipo.20755$$o10.1002/hipo.20755
000140884 999C5 $$2Crossref$$9-- missing cx lookup --$$a10.1007/978-94-024-1088-4_11$$o10.1007/978-94-024-1088-4_11
000140884 999C5 $$2Crossref$$9-- missing cx lookup --$$a10.3390/cells3020288$$o10.3390/cells3020288
000140884 999C5 $$2Crossref$$9-- missing cx lookup --$$a10.1146/annurev-neuro-070815-014006$$o10.1146/annurev-neuro-070815-014006