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@ARTICLE{Fischer:144745,
      author       = {Fischer, A. and Schneider, A. and Wüllner, U.},
      title        = {{E}pigenetik – neue {E}rkenntnisse zum {V}erständnis
                      neurodegenerativer {E}rkrankungen | {E}pigenetic
                      {M}echanisms in {N}eurodegenerative {D}iseases},
      journal      = {Aktuelle Neurologie},
      volume       = {42},
      number       = {07},
      issn         = {0302-4350},
      address      = {Stuttgart},
      publisher    = {Thieme},
      reportid     = {DZNE-2020-00195},
      pages        = {393-401},
      year         = {2015},
      abstract     = {Epigenetics describes the various structures and functions
                      of the DNA-protein matrix (chromatin), which regulate gene
                      expression beyond the DNA sequence. The epigenome determines
                      the differences in the development of genetically identical
                      cells and individuals (mono-zygotic twins). Epigenetic
                      features such as direct biochemical modifications of DNA
                      (methylation) are affected by environmental conditions
                      (famine, early life stress) and are inherited in a modified
                      form, without alteration of the DNA sequence. The analysis
                      of epigenetic processes, in particular DNA methylation,
                      modifications of the DNA-binding proteins, and the functions
                      of the non-coding microRNA, opens an unexpectedly complex
                      field of environment-gene interactions that affect the
                      individual patterns of disease symptoms as well as effects
                      and side effects of many drugs.The study of the epigenome
                      hold the promise of new diagnostic and possibly therapeutic
                      perspectives also for the two most common neurodegenerative
                      diseases, namely Alzheimerʼs and Parkinsonʼs disease.},
      cin          = {AG Wüllner / AG Fischer ; AG Fischer / AG Schneider
                      Göttingen},
      ddc          = {610},
      cid          = {I:(DE-2719)1011302 / I:(DE-2719)1410002 /
                      I:(DE-2719)1440011},
      pnm          = {344 - Clinical and Health Care Research (POF3-344) / 342 -
                      Disease Mechanisms and Model Systems (POF3-342)},
      pid          = {G:(DE-HGF)POF3-344 / G:(DE-HGF)POF3-342},
      typ          = {PUB:(DE-HGF)16},
      doi          = {10.1055/s-0035-1552630},
      url          = {https://pub.dzne.de/record/144745},
}