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@ARTICLE{Leblanc:165175,
      author       = {Leblanc, Pascal and Vorberg, Ina},
      title        = {{V}iruses in neurodegenerative diseases: {M}ore than just
                      suspects in crimes.},
      journal      = {PLoS pathogens},
      volume       = {18},
      number       = {8},
      issn         = {1553-7366},
      address      = {Lawrence, Kan.},
      publisher    = {PLoS},
      reportid     = {DZNE-2022-01477},
      pages        = {e1010670},
      year         = {2022},
      abstract     = {Neurodegenerative diseases (NDs) such as Alzheimer's and
                      Parkinson's disease are fatal neurological diseases that can
                      be of idiopathic, genetic, or even infectious origin, as in
                      the case of transmissible spongiform encephalopathies. The
                      etiological factors that lead to neurodegeneration remain
                      unknown but likely involve a combination of aging, genetic
                      risk factors, and environmental stressors. Accumulating
                      evidence hints at an association of viruses with
                      neurodegenerative disorders and suggests that virus-induced
                      neuroinflammation and perturbation of neuronal protein
                      quality control can be involved in the early steps of
                      disease development. In this review, we focus on emerging
                      evidence for a correlation between NDs and viral infection
                      and discuss how viral manipulations of cellular processes
                      can affect the formation and dissemination of
                      disease-associated protein aggregates.},
      subtyp        = {Review Article},
      keywords     = {Aging / Crime / Humans / Neurodegenerative Diseases / Prion
                      Diseases / Viruses},
      cin          = {AG Vorberg},
      ddc          = {610},
      cid          = {I:(DE-2719)1013004},
      pnm          = {352 - Disease Mechanisms (POF4-352)},
      pid          = {G:(DE-HGF)POF4-352},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:35925897},
      pmc          = {pmc:PMC9352104},
      doi          = {10.1371/journal.ppat.1010670},
      url          = {https://pub.dzne.de/record/165175},
}