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@MISC{Mller:195003,
      author       = {Müller, Stephan A and Lichtenthaler, Stefan},
      title        = {{D}ataset: {C}erebrospinal {F}luid ({CSF}) {P}roteomics of
                      {A}30{P}-α{S} mice},
      publisher    = {PRoteomics IDEntifications Database},
      reportid     = {DZNE-2023-00186},
      year         = {2022},
      abstract     = {Here, we took advantage of well-defined mouse models for
                      α-synucleinopathy (A30P-αS ) to explore proteome changes
                      in the cerebrospinal fluid which are related to these
                      distinct proteopathic lesions. Non-targeted liquid
                      chromatography-mass spectrometry revealed that the majority
                      of proteins that undergo age- and disease-related changes in
                      either mouse model was linked to microglia, and more
                      specifically to previously described disease state-specific
                      microglia transcriptomic signatures. The finding that such
                      transcriptomic changes translate into corresponding protein
                      changes in cerebrospinal fluid is of high clinical
                      relevance, supporting efforts to identify bodily fluid
                      biomarkers that reflect the various functional states of
                      microglial activation in Parkinson’s disease.},
      cin          = {AG Lichtenthaler},
      cid          = {I:(DE-2719)1110006},
      pnm          = {352 - Disease Mechanisms (POF4-352)},
      pid          = {G:(DE-HGF)POF4-352},
      typ          = {PUB:(DE-HGF)32},
      url          = {https://pub.dzne.de/record/195003},
}