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@ARTICLE{Antignano:257681,
      author       = {Antignano, Ignazio and Liu, Yingxiao and Offermann, Nina
                      and Capasso, Melania},
      title        = {{A}ging microglia},
      journal      = {Cellular and molecular life sciences},
      volume       = {80},
      number       = {5},
      issn         = {1420-682X},
      address      = {Cham (ZG)},
      publisher    = {Springer International Publishing AG},
      reportid     = {DZNE-2023-00478},
      pages        = {126},
      year         = {2023},
      abstract     = {Microglia are the tissue-resident macrophage population of
                      the brain, specialized in supporting the CNS environment and
                      protecting it from endogenous and exogenous insults.
                      Nonetheless, their function declines with age, in ways that
                      remain to be fully elucidated. Given the critical role
                      played by microglia in neurodegenerative diseases, a better
                      understanding of the aging microglia phenotype is an
                      essential prerequisite in designing better preventive and
                      therapeutic strategies. In this review, we discuss the most
                      recent literature on microglia in aging, comparing findings
                      in rodent models and human subjects.},
      subtyp        = {Review Article},
      keywords     = {Cellular Senescence / Humans / Microglia / Aging / Brain /
                      Neurodegenerative Diseases / Animals / Oxidative Stress /
                      Signal Transduction / Monocytes / Brain-Gut Axis / Aging
                      (Other) / Human microglia (Other) / Microglia (Other) /
                      Neuroinflammation (Other) / RNAseq (Other) / Senescence
                      (Other)},
      cin          = {AG Capasso},
      ddc          = {610},
      cid          = {I:(DE-2719)1013033},
      pnm          = {351 - Brain Function (POF4-351)},
      pid          = {G:(DE-HGF)POF4-351},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:37081238},
      pmc          = {pmc:PMC10119228},
      doi          = {10.1007/s00018-023-04775-y},
      url          = {https://pub.dzne.de/record/257681},
}