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@ARTICLE{Antignano:257681,
author = {Antignano, Ignazio and Liu, Yingxiao and Offermann, Nina
and Capasso, Melania},
title = {{A}ging microglia},
journal = {Cellular and molecular life sciences},
volume = {80},
number = {5},
issn = {1420-682X},
address = {Cham (ZG)},
publisher = {Springer International Publishing AG},
reportid = {DZNE-2023-00478},
pages = {126},
year = {2023},
abstract = {Microglia are the tissue-resident macrophage population of
the brain, specialized in supporting the CNS environment and
protecting it from endogenous and exogenous insults.
Nonetheless, their function declines with age, in ways that
remain to be fully elucidated. Given the critical role
played by microglia in neurodegenerative diseases, a better
understanding of the aging microglia phenotype is an
essential prerequisite in designing better preventive and
therapeutic strategies. In this review, we discuss the most
recent literature on microglia in aging, comparing findings
in rodent models and human subjects.},
subtyp = {Review Article},
keywords = {Cellular Senescence / Humans / Microglia / Aging / Brain /
Neurodegenerative Diseases / Animals / Oxidative Stress /
Signal Transduction / Monocytes / Brain-Gut Axis / Aging
(Other) / Human microglia (Other) / Microglia (Other) /
Neuroinflammation (Other) / RNAseq (Other) / Senescence
(Other)},
cin = {AG Capasso},
ddc = {610},
cid = {I:(DE-2719)1013033},
pnm = {351 - Brain Function (POF4-351)},
pid = {G:(DE-HGF)POF4-351},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:37081238},
pmc = {pmc:PMC10119228},
doi = {10.1007/s00018-023-04775-y},
url = {https://pub.dzne.de/record/257681},
}