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@ARTICLE{Perez:257689,
      author       = {Perez, Maria Jose and Deleidi, Michela},
      title        = {{N}ew insights into the autophagy-{NAD} axis in brain
                      disease.},
      journal      = {Cell reports},
      volume       = {42},
      number       = {5},
      issn         = {2211-1247},
      address      = {[New York, NY]},
      publisher    = {Elsevier},
      reportid     = {DZNE-2023-00486},
      pages        = {112420},
      year         = {2023},
      abstract     = {Sun et al. demonstrate that defects in autophagy cause
                      nicotinamide adenine dinucleotide (NAD) depletion and
                      neurotoxicity.1 Restoring NAD levels rescues cytotoxicity in
                      autophagy-deficient neurons, providing a potential therapy
                      for neurodegenerative and lysosomal storage diseases
                      associated with autophagy defects.},
      keywords     = {Humans / NAD / Brain Diseases / Neurons / Autophagy / CP:
                      Cell biology (Other) / CP: Metabolism (Other) / NAD (NLM
                      Chemicals)},
      cin          = {AG Deleidi},
      ddc          = {610},
      cid          = {I:(DE-2719)1210011},
      pnm          = {352 - Disease Mechanisms (POF4-352)},
      pid          = {G:(DE-HGF)POF4-352},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:37083331},
      doi          = {10.1016/j.celrep.2023.112420},
      url          = {https://pub.dzne.de/record/257689},
}