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001     257689
005     20240611120549.0
024 7 _ |a 10.1016/j.celrep.2023.112420
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024 7 _ |a 2211-1247
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024 7 _ |a 2639-1856
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037 _ _ |a DZNE-2023-00486
041 _ _ |a English
082 _ _ |a 610
100 1 _ |a Perez, Maria Jose
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245 _ _ |a New insights into the autophagy-NAD axis in brain disease.
260 _ _ |a [New York, NY]
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520 _ _ |a Sun et al. demonstrate that defects in autophagy cause nicotinamide adenine dinucleotide (NAD) depletion and neurotoxicity.1 Restoring NAD levels rescues cytotoxicity in autophagy-deficient neurons, providing a potential therapy for neurodegenerative and lysosomal storage diseases associated with autophagy defects.
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650 _ 7 |a CP: Cell biology
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650 _ 7 |a CP: Metabolism
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650 _ 7 |a NAD
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650 _ 2 |a Humans
|2 MeSH
650 _ 2 |a NAD
|2 MeSH
650 _ 2 |a Brain Diseases
|2 MeSH
650 _ 2 |a Neurons
|2 MeSH
650 _ 2 |a Autophagy
|2 MeSH
700 1 _ |a Deleidi, Michela
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773 _ _ |a 10.1016/j.celrep.2023.112420
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856 4 _ |u https://pub.dzne.de/record/257689/files/DZNE-2023-00486.pdf
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910 1 _ |a Deutsches Zentrum für Neurodegenerative Erkrankungen
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