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@ARTICLE{Hilton:272334,
      author       = {Hilton, Brett J and Griffin, Jarred and Fawcett, James W
                      and Bradke, Frank},
      title        = {{N}euronal maturation and axon regeneration: unfixing
                      circuitry to enable repair.},
      journal      = {Nature reviews / Neuroscience},
      volume       = {25},
      number       = {10},
      issn         = {1471-0048},
      address      = {London},
      publisher    = {Nature Publ. Group},
      reportid     = {DZNE-2024-01153},
      pages        = {649 - 667},
      year         = {2024},
      abstract     = {Mammalian neurons lose the ability to regenerate their
                      central nervous system axons as they mature during embryonic
                      or early postnatal development. Neuronal maturation requires
                      a transformation from a situation in which neuronal
                      components grow and assemble to one in which these
                      components are fixed and involved in the machinery for
                      effective information transmission and computation. To
                      regenerate after injury, neurons need to overcome this fixed
                      state to reactivate their growth programme. A variety of
                      intracellular processes involved in initiating or sustaining
                      neuronal maturation, including the regulation of gene
                      expression, cytoskeletal restructuring and shifts in
                      intracellular trafficking, have been shown to prevent axon
                      regeneration. Understanding these processes will contribute
                      to the identification of targets to promote repair after
                      injury or disease.},
      subtyp        = {Review Article},
      keywords     = {Animals / Nerve Regeneration: physiology / Axons:
                      physiology / Humans / Neurogenesis: physiology / Neurons:
                      physiology},
      cin          = {AG Bradke},
      ddc          = {570},
      cid          = {I:(DE-2719)1013002},
      pnm          = {351 - Brain Function (POF4-351)},
      pid          = {G:(DE-HGF)POF4-351},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:39164450},
      doi          = {10.1038/s41583-024-00849-3},
      url          = {https://pub.dzne.de/record/272334},
}