000279498 001__ 279498
000279498 005__ 20250710100950.0
000279498 037__ $$aDZNE-2025-00825
000279498 1001_ $$0P:(DE-HGF)0$$aStein, Frank$$b0
000279498 245__ $$aDataset: GBA1 interactome for understanding pathological mechanisms
000279498 260__ $$bPRoteomics IDEntifications Database$$c2023
000279498 3367_ $$2BibTeX$$aMISC
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000279498 520__ $$aCompound heterozygous or homozygous GBA1 mutations lead to Gaucher disease. Furthermore, GBA1 mutations are the most frequent risk factor for Parkinson's disease. To get a better understanding of the pathological mechanisms, we generated inducible V5-Flag-Tag, V5-Flag-tagged WT, E326K and L444P mutant Flp-Inâ„¢T-RExâ„¢-HEK293 and performed interatomic analysis.
000279498 536__ $$0G:(DE-HGF)POF4-352$$a352 - Disease Mechanisms (POF4-352)$$cPOF4-352$$fPOF IV$$x0
000279498 7001_ $$0P:(DE-2719)2810385$$aDeleidi, Michela$$b1$$udzne
000279498 7870_ $$0DZNE-2023-00443$$aBaden, Pascale et.al.$$d[London] : Nature Publishing Group UK, 2023$$iRelatedTo$$r$$tGlucocerebrosidase is imported into mitochondria and preserves complex I integrity and energy metabolism.
000279498 8564_ $$uhttps://wwwdev.ebi.ac.uk/pride/archive/projects/PXD032155
000279498 909CO $$ooai:pub.dzne.de:279498$$pVDB
000279498 9101_ $$0I:(DE-588)1065079516$$6P:(DE-2719)2810385$$aDeutsches Zentrum für Neurodegenerative Erkrankungen$$b1$$kDZNE
000279498 9131_ $$0G:(DE-HGF)POF4-352$$1G:(DE-HGF)POF4-350$$2G:(DE-HGF)POF4-300$$3G:(DE-HGF)POF4$$4G:(DE-HGF)POF$$aDE-HGF$$bGesundheit$$lNeurodegenerative Diseases$$vDisease Mechanisms$$x0
000279498 9201_ $$0I:(DE-2719)1210011$$kAG Deleidi$$lMitochondria and Inflammation in Neurodegenerative Diseases$$x0
000279498 980__ $$adataset
000279498 980__ $$aVDB
000279498 980__ $$aI:(DE-2719)1210011
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