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@ARTICLE{Man:281842,
author = {Man, Kevin and Duarte da Silva, Vinicius A and Potemkin,
Nikita and Gabriel, Sarah S and Mason, Teisha and Elmzzahi,
Tarek and De Lima Moreira, Marcela and Su, Chun-Hsi and
Mackay, Laura and Beyer, Marc D and Schröder, Jan and
Gasteiger, Georg and Kallies, Axel},
title = {{S}tem-like tissue-resident memory {T} cells control
functional heterogeneity and reactivation of {T} cell memory
in the intestine.},
journal = {Science immunology},
volume = {10},
number = {112},
issn = {2470-9468},
address = {Washington, DC},
publisher = {AAAS},
reportid = {DZNE-2025-01221},
pages = {eadw1992},
year = {2025},
abstract = {Tissue-resident memory T (TRM) cells provide localized
immunity against intracellular pathogens and cancer. Upon
antigen reencounter, TRM cells differentiate into effector
cells while also giving rise to another generation of memory
cells. Here, we show that intestinal TRM cells that express
the transcriptional regulators TCF1 or ID3 exhibit stem-like
memory properties and are endowed with a superior capacity
to regenerate effector and memory T cells after pathogen
reencounter. Ablation of TCF1 using a TRM cell-specific
mouse model resulted in impaired formation of intestinal TRM
cells, altered their transcriptional heterogeneity, and
increased their differentiation into tissue-confined and
recirculating CX3CR1+ effector cells during recall. TGF-β
and retinoic acid were required for formation and survival
of TCF1- and ID3-expressing TRM cells and restrained their
differentiation into CX3CR1+ effector cells during
reinfection. Thus, stem-like cells control the quality and
recall capacity of TRM cells, thereby contributing to
anamnestic memory responses.},
keywords = {Animals / Immunologic Memory: immunology / Memory T Cells:
immunology / Mice / Mice, Inbred C57BL / Intestines:
immunology / Hepatocyte Nuclear Factor 1-alpha: genetics /
Hepatocyte Nuclear Factor 1-alpha: immunology / Cell
Differentiation: immunology / Inhibitor of Differentiation
Proteins: immunology / Inhibitor of Differentiation
Proteins: genetics / CX3C Chemokine Receptor 1 / Stem Cells:
immunology / Mice, Knockout / T Cell Transcription Factor 1:
genetics / T Cell Transcription Factor 1: immunology /
Hepatocyte Nuclear Factor 1-alpha (NLM Chemicals) / Hnf1a
protein, mouse (NLM Chemicals) / Idb3 protein, mouse (NLM
Chemicals) / Inhibitor of Differentiation Proteins (NLM
Chemicals) / CX3C Chemokine Receptor 1 (NLM Chemicals) / T
Cell Transcription Factor 1 (NLM Chemicals)},
cin = {AG Beyer / PRECISE},
ddc = {610},
cid = {I:(DE-2719)1013035 / I:(DE-2719)1013031},
pnm = {351 - Brain Function (POF4-351) / 352 - Disease Mechanisms
(POF4-352)},
pid = {G:(DE-HGF)POF4-351 / G:(DE-HGF)POF4-352},
experiment = {EXP:(DE-2719)PRECISE-20190321},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41171881},
doi = {10.1126/sciimmunol.adw1992},
url = {https://pub.dzne.de/record/281842},
}